[1] |
Franklin M, Sperry MM, Phillips E, et al. Painful temporomandibular joint overloading induces structural remodeling in the pericellular matrix of that joint's chondrocytes[J]. J Orthop Res, 2021, doi: 10.1002/jor.25050.
|
[2] |
徐高丽,肖芳,霍光.颞下颌关节盘移位后关节的适应性改建[J].口腔颌面外科志,2016,26(1): 61-64.
|
[3] |
Li B, Guan G, Mei L, et al. Pathological mechanism of chondrocytes and the surrounding environment during osteoarthritis of temporomandibular joint[J]. J Cell Mol Med, 2021, 25(11):4902-4911.
|
[4] |
Fang L, Ye Y, Tan X, et al. Overloading stress-induced progressive degeneration and self-repair in condylar cartilage[J]. Ann N Y Acad Sci, 2021, doi: 10.1111/nyas.14606.
|
[5] |
Lekvijittada K, Hosomichi J, Maeda H, et al. Intermittent hypoxia inhibits mandibular cartilage growth with reduced TGF-β and SOX9 expressions in neonatal rats[J]. Sci Rep, 2021, 11(1):1-12.
|
[6] |
Feng Y, Hu S, Liu L, et al. HMGB1 contributes to osteoarthritis of temporomandibular joint by inducing synovial angiogenesis[J]. J Oral Rehabil, 2021, 48(5):551-559.
|
[7] |
徐高丽,吴立立,谷志远,等.缺氧诱导因子-1α信号通路在髁突软骨生长和改建中的作用机制[J].华西口腔医学杂志,2016,34(6): 639-642.
|
[8] |
Kierans SJ, Taylor CT. Regulation of glycolysis by the hypoxia-inducible factor (HIF): Implications for cellular physiology[J]. J Physiol, 2021, 599(1):23-37.
|
[9] |
Fu L, Zhang LW, Zhang X, et al. Roles of oxygen level and hypoxia-inducible factor signaling pathway in cartilage, bone and osteochondral tissue engineering[J]. Biomed Mater, 2021, 16(2):022006.
|
[10] |
Mohammadi B, Esmaeilizade Z, Omrani MD, et al. The effect of co-treating human mesenchymal stem cells with epigallocatechin gallate and hypoxia inducible factor-1 on the expression of RANKL/RANK/OPG signaling pathway, osteogenesis, and angiogenesis genes[J]. Regen Eng Transl Med, 2021, doi: 10.1007/s40883-021-00197-z.
|
[11] |
Taheem DK, Jell G, Gentleman E. Hypoxia Inducible Factor-1α in Osteochondral Tissue Engineering[J]. Tissue Eng Part B Rev, 2020, 26(2):105-115.
|
[12] |
Cramer T, Schipani E, Johnson RS, et al. Expression of VEGF isoforms by epiphyseal chondrocytes during low-oxygen tension is HIF1-α dependent[J]. Osteoarthritis Caetilage, 2004, 12(6):433-439.
|
[13] |
Mino-Oka A, Izawa T, Shinohara T, et al. Roles of hypoxia inducible factor-1α in the temporomandibular joint[J]. Arch Oral Biol, 2017, 73:274-281.
|
[14] |
Li H, Liao L, Hu Y, et al. Identification of type H vessels in mice mandibular condyle[J]. J Dent Res, 2021, 100(9):983-992.
|
[15] |
Yu J, Liang F, Huang H, et al. Effects of loading on chondrocyte hypoxia, HIF-1α and VEGF in the mandibular condylar cartilage of young rats[J]. Orthod Craniofac Res, 2018, 21(1):41-47.
|
[16] |
Fernández-Torres J, Zamudio-Cuevas Y, Martínez-Nava GA, et al. Hypoxia inducible factors (HIFs) in the articular cartilage: A systematic review[J]. Eur Rev Med Pharmacol Sci, 2017, 21(12):2800-2810.
|
[17] |
Pfander D, Cramer T, Schipani E, et al. HIF-1alpha controls extracellular matrix synthesis by epiphyseal chondrocytes[J]. J Cell Sci, 2003, 116(Pt 9):1819-1826.
|
[18] |
Kim CH, Cho YS, Chun YS, et al. Early expression of myocardial HIF-1 alpha in response to mechanical stresses: Regulation by stretch-activated channels and the phosphatidylinositol 3-kinase signaling pathway[J]. Circ Res, 2002, 90(2):E25-33.
|
[19] |
Cheng M, Yi X, Zhou Q. Overexpression of HIF-1 alpha in bone marrow mesenchymal stem cells promote the repair of mandibular condylar osteochondral defect in a rabbit model[J]. J Oral Maxillofac Surg, 2021, 79(2):345.
|
[20] |
Tang Y, Hong C, Cai Y, et al. HIF-1α mediates osteoclast-induced mandibular condyle growth via AMPK signaling[J]. J Dent Res, 2020, 99(12):1377-1386.
|
[21] |
Chen Y, Zhao B, Zhu Y, et al. HIF-1-VEGF-Notch mediates angiogenesis in temporomandibular joint osteoarthritis[J]. Am J Transl Res, 2019, 11(5):2969-2982.
|
[22] |
Kong P, Chen R, Zou FQ, et al. HIF-1α repairs degenerative chondrocyte glycolytic metabolism by the transcriptional regulation of Runx2[J]. Eur Rev Med Pharmacol Sci, 2021, 25(3):1206-1214.
|